Related LncRNAs
ID	lncRNA Name	Disease	Method	Sample	Expression pattern	Dysfunction type	Description	PMID	Source
EL0001	116HG	Prader-Willi syndrome	N/A	N/A	N/A	regulation	Long ncRNA 116HG has been shown to play a role in the development of Prader–Willi syndrome (PWS) (Powell et al., 2013).	24624135	LncRNADisease
EL0556	H19	Prader-Willi syndrome	N/A	N/A	N/A	regulation	Epigenetic deregulation of lncRNAs genes is associated with disease	23791884	LncRNADisease
EL0618	IPW	Prader-Willi syndrome	N/A	N/A	N/A	mutation	Genetic variation in lncRNA genes causes disease and influences susceptibility	23791884	LncRNADisease
EL0618	IPW	Prader-Willi syndrome	N/A	N/A	N/A	expression	Subsequently, we determined that IPW, along?noncoding?RNA?in the critical region of the PWS locus, is a regulator of the DLK1-DIO3 region, as its overexpression in PWS and parthenogenetic iPSCs resulted in downregulation of MEGs in this locus.?	24816254	LncRNADisease
EL0618	IPW	Prader-Willi syndrome	N/A	N/A	N/A	locus	IPW (Imprinted gene in the Prader-Willi syndrome region) was isolated using the direct selection method and yeast artificial chromosomes localized to the deletion region. the mRNA product of IPW may play a role in the imprinting process.	7849716	LncRNADisease
EL0618	IPW	Prader-Willi syndrome	N/A	N/A	N/A	expression	The Prader-Willi syndrome (PWS) is caused by genomic alterations that inactivate imprinted, paternally expressed genes in human chromosome region 15q11-q13. IPW, a paternally expressed gene cloned from this region, is not expressed in individuals with PWS.	9063754	LncRNADisease
EL0885	MKRN3-AS1	Prader-Willi syndrome	N/A	N/A	N/A	epigenetics	ZNF127 and ZNF127AS are imprinted genes that may be associated with some of the clinical features of the polygenic Prader-Willi syndrome.	10196367	LncRNADisease
EL0989	NPAP1	Prader-Willi syndrome	N/A	N/A	N/A	expression	C15orf2 and a novel noncoding transcript from the Prader-Willi/Angelman syndrome region show monoallelic expression in fetal brain.	17337158	LncRNADisease
EL1224	SNHG14	Prader-Willi syndrome	N/A	N/A	N/A	expression	Prader–Willi and Angelman syndromes, due to UBE3A expression, are affected due to the presence of a 20-kb NAT, which shares complementary sequences of UBE3A	23781896	LncRNADisease