LncRNA Information | ||||||
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ID | EL0718 | Name | linc-ITGB1 | Aliases | linc-ITGB1 | |
Species | Homo sapiens | Chromosome | 10 | Start site | 32900319 | |
End site | 33005792 | Chain | minus | Exon NO. | 18 | |
Assembly | Ensembl Release 89 | Class | lincRNA | NCBI accession | N/A | |
Ensembl | ENSG00000150093 | Sequence | N/A |
Disease | |||||||||
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Disease | Method | Sample | Expression pattern | Dysfunction type | Description | PMID | Source | ||
breast cancer | qPCR, Western blot, knockdown, Flow cytometry assay etc. | breast cancer tissue, cell lines (MDA-MB-231, MCF-7, T47D, ZR-75-30, 1590 etc.) | up-regulated | interaction | The expression of linc-ITGB1 was significantly upregulated in both clinical breast cancer tissues and cultured breast cancer cell lines. Linc-ITGB1 depletion caused cell accumulation in the G0/G1 phase. Furthermore, the linc-ITGB1 knockdown decreased the expression of mesenchymal markers N-cadherin and vimentin while increasing the expression of the epithelial marker E-cadherin. Key cell cycle regulators Cdc25C and Cyclin B1 were also decreased by the linc-ITGB1 knockdown. These data suggest that linc-ITGB1 promotes breast cancer progression by inducing cell-cycle arrest and interrupting the epithelial-to-mesenchymal transition (EMT) process. | 26601916 | Lnc2Cancer | ||
Interaction | |||||||||
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Interaction target | Level of interaction | Type of interaction | Description | PMID | Source | ||||
N-cadherin, vimentin, E-cadherin, Cdc25C, Cyclin B1 | RNA-Protein | regulation | The linc-ITGB1 knockdown decreased the expression of mesenchymal markers N-cadherin and vimentin while increasing the expression of the epithelial marker E-cadherin. Key cell cycle regulators Cdc25C and Cyclin B1 were also decreased by the linc-ITGB1 knockdown. | 26601916 | |||||