Disease |
Disease |
Method |
Sample |
Expression pattern |
Dysfunction type |
Description |
PMID |
Source |
nasopharyngeal carcinoma |
knockdown, colony-formation, wound-healing, and transwell assays, cell cycle analysis, western blot analysis |
tumor tissues from 20 patients with nasopharyngeal carcinoma (NPC) as well as from cultured NPC cell lines |
N/A |
expression |
LncRNA, HNF1A-AS potentially regulates NPC tumorigenesis. HNF1A-AS was found to be associated with epithelial to mesenchymal transition. |
26756620 |
|
gastric cancer |
microarray, qPCR etc. |
GC tissue, cell lines (AGS, BGC-823, MKN-45) |
down-regulated |
interaction |
LncRNAs microarray analysis results exhibited that HNF1A-AS1 was downregulated in GCs tissues (mean fold change 2.06, p < 0.05), which was further confirmed by qRT-PCR. The results from qRT-PCR showed that the expression of HNF1A-AS1 was not only downregulated in three GC cell lines (AGS, BGC-823, and MKN-45) relative to that in a normal gastric mucosal epithelial cell line (GES-1), but also decreased in GC tissues relative to that in paired adjacent non-neoplastic tissues (low expression, 94 of 161; low expression rate, 58.38%). Furthermore, low HNF1A-AS1 expression was associated with tumor size/diameter (p = 0.005, multivariate analysis), levels of serum carcinoembryonic antigen (CEA), and carbohydrate antigen 19-9 (CA19-9), and RRM1 expression in tissue samples |
26472090 |
Lnc2Cancer
|
esophageal adenocarcinoma |
microarray, RNA-seq, qPCR, Western bolt, knockdown etc. |
esophageal adenocarcinoma tissue, cell lines (HEEpiC, SKGT-4, OE33 etc.) |
up-regulated |
regulation |
Long non-coding RNA HNF1A-AS1 regulates proliferation and migration in oesophageal adenocarcinoma cells. |
24000294 |
LncRNADisease Lnc2Cancer
|
hepatocellular carcinoma |
N/A |
HCC tissue and cell line |
up-regulated |
N/A |
sponging tumor-suppressive hsa-miR-30b-5p (miR-30b) and de-repressing Bcl-2 |
27084450 |
|
esophageal squamous cell carcinoma |
qPCR etc. |
blood (plasma and serum) |
up-regulated |
expression |
Furthermore, plasma levels of POU3F3, HNF1A-AS1 and SPRY4-IT1 were significantly higher in ESCC patients compared with normal controls.By receiver operating characteristic curve (ROC) analysis, among the three lncRNAs investigated, plasma POU3F3 provided the highest diagnostic performance for detection of ESCC (the area under the ROC curve (AUC), 0.842. |
25608466 |
Lnc2Cancer
|
lung adenocarcinoma |
qPCR, Western blot, knockdown, RIP etc. |
lung adenocarcinoma tissue, cell lines (A549, SPC-A1, H1650) |
up-regulated |
interaction |
HNF1A-AS1 was significantly up-regulated in lung adenocarcinoma tissues compared with corresponding non-tumor tissues, and its expression level was significantly correlated with TNM stage, tumor size, and lymph node metastasis. Moreover, HNF1A-AS1 was determined to promote tumor proliferation and metastasis, both in vitro and in vivo, by regulating cyclin D1, E-cadherin, N-cadherin and β-catenin expression. In addition, the binding of HNF1A-AS1 to DNMT1 may explain its regulation of E-cadherin. |
25863539 |
Lnc2Cancer
|
|