Disease |
Disease |
Method |
Sample |
Expression pattern |
Dysfunction type |
Description |
PMID |
Source |
prostate cancer |
microarray, RNA-seq, qPCR, Northern bolt, knockdown etc. |
prostate cancer tissue, cell lines (LNCaP, CWR22Rv1, PC3 etc.) |
up-regulated |
N/A |
Interestingly, nine out of these thirteen known cancer-related lncRNA showed significantly differential expression between tumor and normal prostate samples. Several lncRNA such as NEAT1, DANCR, HOTTIP, PRINS, and EGOT that have established functions in forming nuclear speckles, in development or in autoimmune disease, but were not previously known to be related to cancer, showed differential expression between tumor and normal prostate samples, suggesting their potential function in prostate cancer. |
23728290 |
Lnc2Cancer
|
Beckwith-Wiedemann syndrome |
N/A |
N/A |
N/A |
locus |
KvDMR1 and/or its associated antisense RNA (KvLQT1-AS) represents an additional imprinting control element or center in the human 11p15.5 and mouse distal 7 imprinted domains. |
10393948 |
LncRNADisease
|
Beckwith-Wiedemann syndrome |
N/A |
N/A |
N/A |
locus |
The LIT1 CpG island can act as a negative regulator in cis for coordinate imprinting at the centromeric domain, thereby suggesting a role for the LIT1 locus in a BWS pathway leading to functional inactivation of p57(KIP2). |
10958646 |
LncRNADisease
|
Beckwith-Wiedemann syndrome |
N/A |
N/A |
N/A |
epigenetics |
The 5'end of the KCNQ1OT1 gene is hypomethylated in the Beckwith-Wiedemann syndrome. |
12136243 |
LncRNADisease
|
Beckwith-Wiedemann syndrome |
N/A |
N/A |
N/A |
N/A |
In vitro fertilization may increase the risk of Beckwith-Wiedemann syndrome related to the abnormal imprinting of the KCN1OT gene. |
12746837 |
LncRNADisease
|
Beckwith-Wiedemann syndrome |
N/A |
N/A |
N/A |
locus |
In the human and mouse BWS imprinting regions, two major elements for regulation of imprinted gene expression have been identified athe imprinting centers IC1 and IC2.IC2 appears to be the promoter of the paternally expressed probably noncoding transcript. |
15590939 |
LncRNADisease
|
Beckwith-Wiedemann syndrome |
N/A |
N/A |
N/A |
N/A |
LIT1 (KCNQ1OT1) may play a role in Beckwith-Wiedemann syndrome. |
15888726 |
LncRNADisease
|
Beckwith-Wiedemann syndrome |
N/A |
N/A |
N/A |
epigenetics |
In Beckwith-Wiedemann syndrome (BWS), approximately 50% of patients show loss of DNA methylation accompanied by loss of histone H3 Lys9 dimethylation on maternal KCNQ1OT-DMR, namely an imprinting disruption, leading to diminished expression of CDKN1C. |
16575194 |
LncRNADisease
|
colorectal cancer |
N/A |
N/A |
N/A |
expression |
Recent studies have linked their mis-expression to diverse cancers (ANRIL: prostate cancer, XIST: female cancers, HOTAIR: breast cancer, KCNQ1OT4: colorectal cancer). |
23660942 |
LncRNADisease
|
Beckwith-Wiedemann syndrome |
N/A |
N/A |
N/A |
regulation |
Epigenetic deregulation of lncRNAs genes is associated with disease |
23791884 |
LncRNADisease
|
kidney cancer |
N/A |
N/A |
N/A |
regulation |
Oncogene |
24373479 |
LncRNADisease
|
acute myocardial infarction |
N/A |
N/A |
N/A |
N/A |
Level of KCNQ1OT1 was higher in patients with MI than in healthy volunteers (P<0.01);Patients with ST-segment-elevation MI had lower levels of KCNQ1OT1 (P<0.001)when compared with patients with non-ST-segment-elevation |
25035150 |
LncRNADisease
|
colorectal cancer |
overexpression |
colorectal cancer cells |
up-regulated |
N/A |
β-catenin can promote KCNQ1OT1 transcription through direct binding to the KCNQ1OT1 promoter |
26868975 |
|
hepatocelluar carcinoma |
qPCR etc. |
HCC tissue |
differential expression |
mutation |
A novel tetranucleotide repeat polymorphism within KCNQ1OT1 confers risk for hepatocellular carcinoma. |
23984860 |
LncRNADisease Lnc2Cancer
|
colorectal cancer |
qPCR, FISH etc. |
CRC tissue, cell lines (FBS, DLD-1, HCT-15 etc.) |
differential expression |
epigenetics |
epigenetic disruption |
16965397 |
LncRNADisease Lnc2Cancer
|
breast cancer |
RNA CISH etc. |
breast cancer tissue |
up-regulated |
expression |
HOTAIR, H19 and KCNQ1OT1 had significantly higher expression levels in IBC than NA, and HOTAIR and H19 were both expressed more strongly in IBC than in DCIS tissues. |
26323944 |
Lnc2Cancer
|
|