| LncRNA Information | ||||||
|---|---|---|---|---|---|---|
| ID | EL0615 | Name | IPS1 | Aliases | N/A | |
| Species | Nicotiana benthamiana | Chromosome | N/A | Start site | N/A | |
| End site | N/A | Chain | N/A | Exon NO. | N/A | |
| Assembly | N/A | Class | lincRNA | NCBI accession | N/A | |
| Ensembl | N/A | Sequence | N/A | |||
| Function (not disease relevant) | |||||||||
|---|---|---|---|---|---|---|---|---|---|
| Methods | Sample/condition | Expression pattern | Dysfunction type | Description | PMID | Source | |||
| Northern blot/qRT-PCR | phosphate balance | up-regulated | N/A | The effect of miR-399 on PHO2: GFP was suppressed by simultaneous expression of IPS1, and a mutant form of IPS1 with reduced complementarity to miR-399 did not affect miR-399-mediated silencing of PHO2: GFP. Finally, we reconstituted the interaction between IPS1, miR-399 and PHO2 in a system in which complementary and compensatory mutations were introduced in all three components. | 17643101 | PLNlncRbase | |||
| Interaction | |||||||||
|---|---|---|---|---|---|---|---|---|---|
| Interaction target | Level of interaction | Type of interaction | Description | PMID | Source | ||||
| PHO2 | RNA-RNA | regulation | IPS1 overexpression results in increased accumulation of the miR-399 target PHO2 mRNA and, concomitantly, in reduced shoot Pi content. | 17643101 | PLNlncRbase | ||||